While timely reperfusion can reduce more extensive brain tissue injury by salvaging reversibly damaged penumbra, late recanalization also carries the risk of causing additional and substantial brain damage, such as ischemia-reperfusion injury, compared with no revascularization. The reperfusion status after thrombolysis within the DWI lesion territory and its relationship with HT is unclear, leaving the details regarding post-treatment care unspecified. In this study, we aim to quantify the volume of reperfusion into severely damaged brain tissue and investigate its predictive value for impending parenchymal hematoma following recanalization.
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