Perinatal hypoxic-ischemic encephalopathy (HIE) is one of the most common causes of severe, long-term neurologic deficits in children. Up to 40% of infants with HIE who present minimal to no abnormality on structure MRI may still have manifest neurological deficits in later life. In this study, we hypothesize that functional connectivity (FC) of the brain in infants with HIE at term may be altered and that resting-state functional MRI (rs-fMRI) may provide deeper insights into the altered nature of the brain networks, especially the motor network and the thalamocortical network in infants with HIE.
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